SOD1/Rag2 Mice with Low Copy Number of SOD1 Gene as a New Long-Living Immunodeficient Model of ALS | Scientific Reports
Neuron-Specific Expression of Mutant Superoxide Dismutase Is Sufficient to Induce Amyotrophic Lateral Sclerosis in Transgenic Mice | Journal of Neuroscience
SOD1-G93A Mouse Model - QPS Neuropharmacology
SOD1 aggregation in ALS mice shows simplistic test tube behavior | PNAS
![PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/cf77f873405b248b7c9948af453d095fddb3ca81/3-Figure2-1.png "PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar")
PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar
004435 - SOD1-G93A Strain Details
Characterization of somatosensory neuron involvement in the SOD1G93A mouse model | Scientific Reports
Decreasing SOD1 in muscle does not improve grip strength in SOD1 G93A... | Download Scientific Diagram
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar
Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS) | PLOS ONE
Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse model of Amyotrophic Lateral Sclerosis | Semantic Scholar
Oral Treatment with CuII(atsm) Increases Mutant SOD1 In Vivo but Protects Motor Neurons and Improves the Phenotype of a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis | Journal of Neuroscience
Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits Disease Phenotype Progression in an ALS Mouse Model
Neuroprotective effects of siRNAp11 in the SOD1-G93A mouse model of... | Download Scientific Diagram
IJMS | Free Full-Text | Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice
Overexpression of MT-I in SOD1 G93A mice ameliorates ALS-like... | Download Scientific Diagram
002726 - SOD1-G93A Strain Details
Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology | PNAS
Therapeutic effects of immunization with mutant superoxide dismutase in mice models of amyotrophic lateral sclerosis | PNAS
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports
ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions: Neuron
Generation and analysis of innovative genomically humanized knockin SOD1, TARDBP (TDP-43), and FUS mouse models - ScienceDirect
